Roles of p38 MAP kinase for VEGF synthesis stimulated by TGF-β and 1,25-dihydroxyvitamin D_3 in aortic smooth muscle cells 1) Involvement of p38 MAP kinase in TGF-β-stimulated VEGF synthesis in aortic smooth muscle cells 2) 1,25-Dihydroxyvitamin D_3 stimulates vascular endothelial growth factor release in aortic smooth muscle cells: role of p38 mitogen-activated protein kinase

present study has provided immunohistochemical evidence of the occurrence of VEGF in subsets of peptideamine endocrine and in an exocrine cell type of normal adult rats, namely, the

In contrast, ADAMTS-1 –/– mice exhibit accumulation of excessive fibrous tissue and deformity of smooth muscle cells at the ureteropelvic junction and ureter, which could lead to

Pim-3, a proto-oncogene with serine ⁄ threonine kinase activity, is aberrantly expressed in human pancreatic cancer and phosphorylates Bad to block Bad-mediated apoptosis in

　Quantitative analysis by real-time Reverse transcription-polymerase chain reaction （RT-PCR） of chronological change in the expression of hepatocyte growth factor （HGF）,

However, VPA prevented the morphological changes characteristic for activation and inhibited the expres- sion of collagen type 1 α 1 (COL1A1) and TGF- β 1 in activated LI90 cells

Treatment with ONO-1301 increased hepatic HGF mRNA expression, but decreased the expressions of TGF-β1, connective tissue growth factor, α-smooth muscle actin, and type-I and

GA or an aqueous extract of Schisandra chinensis induced not only endothelium- dependent but also endothelium-independent vascular relaxation in isolated thoracic aorta

These findings further suggest that CD45 + /ColI + may contribute to kidney fibrosis by producing MCP-1/CCL2 and TGF-beta, which may be responsible for chronic